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Assuming a preferential central action of GLP1/E, we conducted label-free, quantitative proteomic analyses in the hypothalamus of the two models of PCOS, after chronic treatment with the di-agonist, as a means to disclose putative pathways for the metabolic actions of the di-agonist, and the basis for the partially differential responses between the two models of PCOS, which diverge also in terms of phenotypic presentation

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Why anorexia may go undiagnosed Media portrayals of disordered eating often focus on anorexia nervosa (AN), a psychiatric disorder marked by severe food restriction, intense fear of weight gain, and dangerously low body weight

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