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Additional file 2: Fig

How your body is processing hormones, medications, and metabolic shifts

These effects are thought to result from combined modulation of hypothalamic appetite-regulating pathways and mesolimbic reward circuits, as shown in studies using pharmacological antagonists and viral knockdown techniques targeting GLP-1Rs

We also developed a method of attaching fatty acids to GLP-1 and staying in the body for a long time

Glutamine triggers and potentiates glucagon-like peptide-1 secretion by raising cytosolic Ca2 + and cAMP

The M2b phenotype can be induced by stimulation with immune complexes (ICs), toll-like receptor (TLR) agonists, or IL-1 receptor ligands
