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Jastreboff and colleagues reported phase 2 clinical-study findings for the LY3437943 program, while earlier discovery and pharmacology data from Coskun et al

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In conclusion, GLP-1RAs can inhibit chondrocyte ferroptosis and alleviate the progress of RA similar to that of C-176 by inhibiting cGAS-STING signaling pathway activation, thereby reducing ROS production and inhibiting ferroptosis(Fig

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While GLP-1RAs demonstrate immunomodulatory potential relevant to SS pathogenesis through their effects on key immune cell populations, direct experimental evidence characterizing their impact on SS-specific immune dysregulation remains strikingly limited