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The increase in the bile acid pool may then stimulate TGR5 bile acid receptors on the L cell [62, 67], causing an increase in GLP-1 secretion via mitochondrial oxidative phosphorylation and calcium influx [67]
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The molecule does not change, but its economic status does
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gastric emptying is delayed in 30% to 50% of individuals, and intragastric meal distribution is also frequently abnormal (37)
PMID: ncbi
