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Tregs are necessary to suppress the chronic inflammation in T2D and to fight insulin resistance, atherosclerosis, and damage to target organs, as in kidney disease (5, 7) ( Figure 1 )

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These findings were substantiated by MS-based proteomics analyses in which we observed a more pronounced response to treatment with GLP-1MK-801 compared with either monotherapy (Fig

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doi: 10.2147/PTT.S485061