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GLP-1 can directly bind to the receptors on the surface of -cells in the pancreas, inhibiting the secretion of glucagon from these cells

In addition to weight reduction, which may increase with longer treatment duration, GIP agonism appears to enhance insulin sensitivity, blood flow, and adipose tissue storage capacity32 (Table 2)

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Abbreviations 5-HIAA: 5-hydroxindile acetic acid 5-HT: 5-hydroxytryptamine AgRP: Agouti-related protein ARH: Arcuate nucleus of the hypothalamus BMI: Body mass index BNST: Bed nucleus of the stria terminalis cAMP: Cyclic adenosine monophosphate d-fen: Dexfenfluramine DRN: Dorsal raphe nucleus FDA: Food and drug administration GABA: Gamma-aminobutyric acid IPSC: Inhibitory post-synaptic current LepR: Leptin receptor LH: Lateral hypothalamus Mc4R: Melanocortin 4 receptors NTS: Nucleus of the solitary tract OX1R: Orexin receptor 1 OX2R: Orexin receptor 2 POMC: Proopiomelanocortin PPG: Preproglucagon PVH: Paraventricular hypothalamic nucleus SERT: Serotonin transporter SSRI: Selective serotonin reuptake inhibitor TPH: Tryptophan hydroxylase Vgat: Vesicular GABA transporters VGLUT: Vesicular glutamate transporters VMH: Ventromedial nucleus of the hypothalamus VTA: Ventral tegmental area WHO: World health organization References Health W, Organization

Beyond their insulinotropic effects, GLP-1R and GIPR signaling also influences lipid metabolism by modulating hepatic lipid handling and adipose tissue function