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In an additional scenario, reduced ERK1/2 and MAPK activity hindered muscle glucose transport and caused insulin resistance (IR) [42]

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These categories differ in the magnitude and direction of receptor activation, enabling tailored therapeutic applications depending on disease mechanisms and required biological responses

It was developed by modifying the natural GLP-1 molecule to: Promote albumin binding: This extends circulation time by slowing renal clearance Resist DPP-4 degradation: Structural modifications protect it from the enzyme that breaks down natural GLP-1 Semaglutide selectively binds to and activates the GLP-1 receptor, triggering the same beneficial downstream effects as natural GLP-1but with sustained activity over days rather than minutes

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