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These benefits were seen despite comparable weight loss and glycemic control, indicating that the effect on cognitive function is independent of the effect on weight and hyperglycemia [59]

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Given what we know about how GLP-1s work on appetite suppression, could the same mechanism fuel an eating disorder in one person while finally treating it in another

We can see that the rate of fasting blood glucose decline in the Low group is slightly slower than that in the High group, but it also maintained at a lower level with the High group at the end of the experiment (Figures 1C, D)
