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Description
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Evidence shows that the strongest genetic risk factor for late-onset AD is the E4 allele of the cholesterol transporter APOE (APOE4), besides, APOE4 could promote amyloid aggregation and impair clearance from the brain directly binding to A (63)

Preclinical studies have demonstrated that GLP-1R agonists can reduce the accumulation of amyloid plaques, a hallmark of Alzheimer's disease, and improve synaptic function

How is the Opportunistic Rise of the Middle East and Africa in the GLP-1 Market
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