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Mutations in the amyloid precursor protein (APP) gene and presenilin genes (PSEN1 and PSEN2) are associated with the production of abnormal amyloid-beta (A) peptides, leading to amyloid plaque formation in the brain

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While traditional GLP-1 agonists have long been the standard for studying weight-related signaling, the addition of GIP receptor activation appears to modulate the adipocyte environment differently

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