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Similarly, semaglutide ameliorates doxorubicin-induced cardiotoxicity by reducing BNIP3-mediated mitochondrial dysfunction through the PI3K/AKT pathway, enhancing mitochondrial function and reducing cardiac injury (Li et al., 2024), potentiating its clinical application in cardiovascular diseases

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This finding is important for developing effective anti-doping tests, as its often the metabolites of a substance, rather than the substance itself, that are detected in biological samples

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