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Secukinumab and Bimekizumab work to reduce lesion counts and inflammation by blocking IL-17A and/or IL-17F, which drive keratinocytes hyperproliferation, the neutrophils recruitment, and chronic inflammation [232,233,234] (Fig
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These substances could enhance glucose regulation by focusing on inflammation and offer additional advantages in addressing coexisting inflammatory disorders
doi: 10.1182/blood-2012-05-423186
