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GIP promotes -cell proliferation and survival through the PI3K/Akt and MAPK/ERK pathways (36,37), while inhibiting apoptosis via regulation of Bcl-2 and Bax expression (38,39)

These molecules influence: GLP-1 and PYY secretion insulin sensitivity mitochondrial energy pathways fat oxidation inflammation modulation appetite and metabolic rhythm Scientific Reference 3: Koh et al., Cell (2016) This high-impact review explains SCFAs mechanistic roles in host physiology: signalling through receptors (e.g., FFAR2/FFAR3), epigenetic modulation, immunegut interactions, and metabolic regulation

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Muscle preservation: why the combination matters for body composition This is the strongest argument for combining keto with GLP-1 therapy

Depot-Specific Effects of Semaglutide on Adipose Tissue To enhance clarity and avoid repetition in the presentation of mechanistic evidence, Figure 3 and Table 1 are intentionally designed to serve complementary roles