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63.8%), lower rates of chronic kidney disease (10.3% vs

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Indeed, nodose GLP-1R lentivirus-mediated knockdown increases postprandial glycemia 128 , and Glp1r Phox2b/ mice, which exhibit selective knockout of GLP-1R in the nodose ganglia, midbrain, and hindbrain, exhibit increased fasting levels of glucose, both highlighting a role for GLP-1 vagal afferent signaling in mediating glucose homeostasis 127

It is noteworthy that the effect of brain GLP-1 on insulin action and vascular blood flow is clearly analyzed in animals, but have never been mentioned in relation to humans

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