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The mechanism involves enhanced insulin secretion from pancreatic -cells only when glucose levels are elevated

DPP-4 inhibitors degrade certain entero-hormones (e.g., glucose-dependent insulinotropic peptide (GIP) and glucagon-like peptide-1 (GLP-1)), or incretins, which are primarily produced within minutes [9]

Critical Role of GLP-1 in Symptomatic Post-Bariatric Hypoglycemia

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adding a weight-loss medication can shift this balance

DPP4 Inhibitors and Mesenteric Arteries Mechanistic studies have been conducted to investigate whether the effects of DPP4 inhibitors on glucose-induced endothelial impairment are associated or independent of GLP-1/GLP-1R