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In healthy subjects, oral glucose triggers a stronger insulin secretion response than intravenous glucose because of the secretion of incretin from the gastrointestinal tract, known as the incretin effect. Clinically, incretin-based drugs include glucagon-like peptide 1 receptor agonists (GLP-1 RA) (such as liraglutide) and dipeptidyl peptidase 4 inhibitors (DPP-4i) (such as vildagliptin)

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It would be important to know more of the demographics of the afflicted populations

This is one of the most misleading things about how these patches are marketed

Dual or triple agonists, for example, GLP-1/GIP/glucagon, have great potential as they have already shown a better therapeutic window in T2DM, as compared to a GLP-1 agonist alone

The research-grade versus pharmaceutical-grade distinction becomes critical here