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Pro-inflammatory cytokines, activation of glial cells, and high levels of reactive oxygen species production (ROS) are known to increase nerve damage and trigger pain signaling pathways [5]

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This is particularly important as online prescriptions are not regulated

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LL-37 can also bind bacterial lipopolysaccharide (LPS) and block its interaction with CD14/TLR4, reducing endotoxin-triggered TNF release and neutrophil apoptosis [2]

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