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GLP-1 signaling has been shown to inhibit activation of the NLRP3 inflammasome, a key driver of innate immune-mediated inflammation, and to enhance Treg activity, suggesting a broader role in restoring immune tolerance [26,27]

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By the time Knudsen returned from maternity leave in 1994, all of her former colleagues had left and the future of the GLP-1 project looked bleak