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Research has established that ATF4 directly induces the transcription of PDE4D, a phosphodiesterase that degrades intracellular cAMP, thereby blunting the signaling fidelity of the GLP-1R (Lee et al., 2023)

GLP-1 induces FNDC5 expression in pancreatic -cells via CREB promoting the conversion of WAT to BAT and thereby enhancing energy metabolism [1]

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LQT1 (30-40% of cases) Mutation: loss of function in potassium channel gene KCNQ1 ECG: broad-based T wave Trigger: activity, adrenaline, exercise BB responsiveness: +++ (nadolol or propranolol) LQT2 (second most common) Mutation: loss of function in potassium channel gene KCNH2 ECG: notched T wave Trigger: auditory (alarm clock), post-partum BB responsiveness: ++ LQT3 Mutation: gain of function or leakiness of sodium channel SCN5A (note: BrS is due to loss of function in the same gene) ECG: normal T wave after prolonged isoelectric ST segment Trigger: none, but typically happens during rest BB responsiveness: + (propranolol)