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Various GLP-1RAs have been demonstrated to have significant benefits in in vitro and in vivo models of neurodegenerative diseases through modulating a variety of pathogenic mechanisms, including neuroinflammation, autophagy, mitochondrial dysfunction, and the abnormal phosphorylation of pathognomonic proteins
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those having a BMI equal to or greater than 25 kg/m2 but less than 30 kg/m2, prevalence more than doubles to 4 billion1
