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Interestingly, exogenously administered GIP and GLP-1 seem to access the brain predominately via leaks in the bloodbrain barrier, where the underlying neuronal tissue has dense expression of their receptors [11,12,13, 17] (Fig

However, a recent study using immunocytochemistry to detect GLP-1 receptors described such receptors not only on human C cells but also in some, but not all, follicular cells and in some papillary thyroid carcinomas, indicating a potential of GLP-1 receptor stimulation to have an influence on the growth rate of other thyroid cancer types as well (7)

Drug development for non-alcoholic steatohepatitis (NASH), also known as metabolic dysfunction-associated steatohepatitis (MASH), has been surprisingly challenging 1

These changes to normal gastrointestinal physiology explain why diarrhea (defined as three or more loose or watery stools per day) and other gastrointestinal symptomsincluding nausea, vomiting, constipation, and abdominal painare among the most frequently reported side effects