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The REWIND trial included patients with hemoglobin A1c levels of up to 9.5%, thus the influence of dulaglutide on glycemic control may underlie the reduced risk of cognitive dysfunction

I felt a bit of fatigue at this point but persisted with the same slightly increased dose for week 4

GCG agonism provides direct hepatic effects by inhibiting lipogenesis and promoting fat oxidation while augmenting energy expenditure

This is where strategy still matters

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It is caused by genetic mutations that lead to a deficiency of the glucocerebrosidase (GCase) enzyme