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More recently, metabolic cage studies have confirmed that the increased oxygen consumption and energy expenditure effect of OXM is mediated through glucagon receptor signalling (39, 40)
At that point, if theyd like to proceed with the GLP1 and they meet criteria, then we roll with it and we consider it harm reduction
GLP-1 users exhibited a numerically higher risk for kidney cancer, but the difference did not reach statistical significance (HR = 1.38
Who Should Not Take GLP-1 Agonists
