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Thats where it gets a little complex, he responds

GLP1 receptor agonist protects palmitate-induced insulin resistance in skeletal muscle cells by up-regulating sestrin2 to promote autophagy

Activation of STING triggers a signaling cascade involving TBK1, IRF3, and NF-B, leading to the production of pro-inflammatory cytokines and further endothelial damage

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It can be argued that the possible hypoglycemic effect is due to concomitant use of other therapies that cause hypoglycemia (eg, sulphonylurea) and it was demonstrated in clinical trials studying a combination of a GLP-1 receptor agonist and a sulphonylurea that the risk of hypoglycemia was higher when compared to placebo