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This reduction in oxidative stress also helps preserve the integrity and function of cholinergic neurons, thereby supporting acetylcholine production [41,42]

Nature Communications, 12(1), 1-11

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Taken together, these results indicate that modulation of direct GLP-1R-cholesterol interactions is a valid strategy to fine-tune beta cell incretin receptor responses, providing a new avenue for further investigation of novel ways to improve GLP-1R signalling outputs

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