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These results suggest that exendin-4-induced suppression may be mediated by a distinct Gs/cAMP-PKA/IP3/Ca2+/CaM/CaMKII signaling pathway, following the activation of GLP-1Rs.

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Recent observations and emerging research have raised concerns about increased tendon injuries and declining bone health in some individuals using GLP-1 therapies

Semaglutide exposure was associated with a higher incidence rate (0228 vs

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