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Therefore, germline loss-of-function variants in the GSC subunits genes can directly trigger the clinical manifestation of HS by compromising Notch-mediated follicular integrity and immune homeostasis
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It also appears to enhance growth of cells in the pancreas, which are the sites of insulin production
doi: 10.1523/JNEUROSCI.5520-03.2004
PMID 28687413
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