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Gene expression analysis to identify mechanisms underlying heart failure susceptibility in mice and humans

Therefore, the activation of the IGF-2/IGF-1R axis may contribute to -cell survival in response to GLP-1, and importantly, the increased IGF-2 secretion may be essential for triggering this response, as also observed in embryonic development (41)

All participants received treatment for at least 52 weeks, with a maximum treatment duration of 104 weeks or until the study was completed to collect and evaluate major adverse cardiovascular events (MACE)

For the majority of practitioners, dosing will begin with a lower amount gradually rising to the maintenance amount