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No matter what weve done, weve not been able to nail precisely why they are resistant. Unexpected resistance The researchers focused on two variants that disrupt an enzyme called PAM (peptidyl-glycine alpha-amidating monooxygenase), which activates several hormones, including GLP-1

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While AT1R mediates classic ANG II actions such as vasoconstriction, aldosterone release, and sodium and water retention, ANG II signaling through AT2R has opposite effects and may represent a protective mechanism against an overstimulation of AT1R (21)