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With NO bioavailability maintained, endothelium-mediated vasorelaxation was preserved and vascular remodeling and fibrosis were prevented (Helmstadter et al

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Stroke and GLP-1RA Putative mechanisms linking GLP-1R activation to reduced rates of stroke are still emerging but are likely multifactorial with indirect effects on platelet aggregation

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The GLP-1R agonist had beneficial effects on systolic and diastolic blood pressure, plasma concentrations of cholesterol, and glycemic control, but did not have a significant impact on plasma concentrations of liver enzymes