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Because Glp1r mRNA is also expressed in the human OB 53 , it is intriguing to explore in future studies whether intranasal administration of GLP-1 agonists 54 could potentially exert their effects by acting in the OB (a site of persistent sensitivity to GLP-1 in obesity according to our data)

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Imagine, instead of waiting until somebody develops type two diabetes and 40 BMI, we catch them at a 25 BMI and put them on a low dose so that it prevents weight gain in the first place and complications that arise in that weight gain

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