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Moreover, SEMA mitigated the TAA-induced histopathological changes and reduced the content of -smooth muscle actin (-SMA)

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It represents a clinically significant manifestation of diabetic autonomic neuropathy and arises from a complex interplay of sustained hyperglycemia, oxidative and nitrosative stress, advanced glycation end-product accumulation, impaired insulin/IGF-1 signaling, lipid dysmetabolism, and inflammatory activation

Following nucleation (and a subsequent desorption step), nucleated droplets grow rapidly, ultimately resulting