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Retatrutide, a not endogenously occurring peptide, stimulated the glucagon receptor (GCGR), the glucose-dependent insulinotropic polypeptide (GIP) receptor (GIPR), and the glucagon-like peptide-1 receptor (GLP-1R) in cell cultures

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There are distinct alterations in metabolic pathways found in serum, urine and cerebrospinal fluid, that resolve following disease remission

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