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MEIS1 and Restless Legs Syndrome: A Comprehensive Review. Frontiers in Neurology, vol
As the surgical restructuring of the gastrointestinal tract represents a mucosal assault that needs to be repaired, one can speculatively suggest that gastric epithelial cell renewal would be engaged into a new endocrine cell differentiation program

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Numerous pharmacological treatments seek to disrupt these harmful connections, with a considerable number focusing on the incretin hormonal pathways and appetite regulation
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This process is accompanied by the activation of protein kinase C (PKC) and the production of intracellular Ca 2+ influx through IP3 receptors