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These increases began from day one all the way through day 60 [R]

These cohorts are evidence but the design constraints are significant and need to be stated plainly: The studies are not blinded, randomized, placebo-controlled trials by Western regulatory standards

Light flaking or peeling may occur over the next few days as the skin renews itself

Possible causative mechanisms of CNS inflammation and neuroglial reactivity include direct effects through injury or infection of the brain, reactivation of endogenous microbial reservoirs in CNS cells, autoimmune reactivity with specific neural, glial, or immune system targets, repetitive mechanical strain, cerebrovascular hypertension, cerebral hypoperfusion and/or ischemia, recognition of danger-associated molecular patterns (DAMP), vagal dysfunction, norepinephrine or angiotensin II overload, or, generally, exposure to chronic stress (VanElzakker, 2013

ERK/MAPK is essential for endogenous neuroprotection in SCN2.2 Cells

Banks et al