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Many patients experience: Delayed gastric emptying Severe nausea Bloating Early satiety Semaglutides mechanism includes: Slowing gastric emptying Reducing GI motility Increasing satiety via vagal pathways Clinical observations also increasingly report: Worsening nausea Increased bloating Vomiting Gastroparesis-like symptoms So, while potentially beneficial in metabolic disease or overweight patients, in individuals already predisposed to gastrointestinal dysmotility, autonomic dysfunction, and vagal dysregulation, these effects can be extremely problematic and counterproductive

The weight medication picture is something weve looked at in relation to fibromyalgia and weight gain and the specific question of weight changes in EDS before, and its a complicated space

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If you notice increased nausea, vomiting, or hypoglycemic symptoms after drinking, stop consuming alcohol and contact your healthcare provider

Thus, dual activation of GIP and GLP-1 receptors may have more significant metabolic enhancement effects than activating either

You can argue the good or bad of that- Rohan Goswami: Are you going to get behind it like Michael Dell has