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This makes it a "dual-agonist." So, while it does the same job as a GLP-1 drug like semaglutide, it has an extra layer of action on the GIP pathway, which is what makes it unique

The upstream part in your body of how that force becomes that signal, even by people who study this for a living, tell us we don't quite know

Some people choose to stay at 1.7 mg if theyre getting good results and dont want to risk increased side effects from the higher dose

These are the same screening questions any responsible semaglutide prescriber should ask

Everyone at LIVation always takes their time and always asks how I am doing and if I have any questions

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