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In addition to aiding in K + ATP channel inhibition, GLP-1R signalling also inhibits a compensatory repolarization event mediated by the opening of voltage-dependent K + channels (K v )

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This narrow focus, however, also restricts our understanding of potential sex differences in GLP-1's role in AN pathology, as it overlooks biological and neurodevelopmental factors that may present differently in males and across age groups

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It induces metabolic reprogramming in response to hypoxia and growth factor signalling 18, 19 , partly by promoting transcriptional activation of glycolytic genes 20

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