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Conclusion Our study identifies OGlcNAcylation at Thr83 as a key modification in obesitydriven tumor progression, facilitating EIF3H recruitment, YAP stabilization, and tumorenhancing functions

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doi: 10.1177/1403494810387965 [DOI] [PubMed] [Google Scholar] 21

7, July 2019, pp

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Glucagon acting at the GLP-1 receptor contributes to -cell regeneration induced by glucagon receptor antagonism in diabetic mice