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Hair loss during semaglutide treatment often stems from rapid weight loss and nutrient shifts, not the medication itself
The authors show that GIPR activation enhances entry of GLP-1R agonists into the brain, and this process depends on oligodendrocyte GIPR signaling
Glucagon-like peptide-1 (GLP-1)is an incretin hormonealso classified as a gut-derived peptidesecreted by intestinal L-cells in response to nutrient ingestion, particularly carbohydrates and fats
