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Linking cellular energy state to atrial fibrillation pathogenesis: potential role of adenosine monophosphate-activated protein kinase

Think of it as an outpouching of the wall of the oesophagus which forms a cul-de-sac

Smits MM et al

[2] The medication works by enhancing glucose-dependent insulin secretion, suppressing glucagon release, slowing gastric emptying, and reducing appetite through central nervous system pathways

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