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A receptor-independent stimulation of GLP-1 release from GLUTag cells has been reported for oleic acid, which stimulates GLP-1 release by uncoupling oxidative phosphorylation and, hence, indirectly stimulating glycolysis with the resulting activation of the mechanism alluded to above [301]
2,3 In addition to these mechanisms, GLP-1 RAs also delay gastric emptying, thereby contributing to enhanced postprandial glucose regulation and increased satiety, ultimately supporting reduced caloric intake and weight management
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