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Moreover, impaired autophagy is associated with sustained inflammation in tissues and may contribute to the pathogenesis of chronic inflammation (76)

Author: Jason Kritzer and James Cullen

However, some scholars have proposed that the effect of GLP-1 in the liver is indirectly mediated by multiple mechanisms, such as reducing fat absorption or inhibiting fat cells through acyl-CoA synthetase long chain family member 1 (ACSL1), CCAAT/enhancer binding protein (C/EBP), peroxisome proliferator-activated receptor- (PPAR), and sterol regulatory element binding protein-1c (SREBP-1c) pathways to achieve the purpose of reducing liver lipids [38]

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References: Baggio LL, Drucker DJ (2007): Biology of incretins: GLP-1 and GIP

[DOI] [PMC free article] [PubMed] [Google Scholar] 915.Neary N.M., Small C.J., Druce M.R., Park A.J., Ellis S.M., Semjonous N.M