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HFpEF and DM share common pathophysiological mechanisms, including systemic inflammation, myocardial fibrosis, endothelial dysfunction, and insulin resistance [3]

The table below summarizes each GLP-1 medication's active ingredient and what the U.S

CONCLUSION The clinical and preclinical data presented at the ACR Convergence 2025 show both the therapeutic potential and the scientific complexity of GLP-1 agonists in OA

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To establish the PPAR-knockout (KO) cell line, the CRISPR/Cas9 system was employed
