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Centrally, semaglutide engages brainstem vomiting centres: it can access circumventricular regions like the area postrema and activate GLP-1 receptors in the dorsal vagal complex (area postrema and nucleus tractus solitarius), directly stimulating nausea and emesis pathways [60,61,62]

Br J Clin Pharmacol 2018;84:56878

Importantly, repeated daily intrathecal injections of morroniside over seven days did not result in the development of tolerance to its anti-allodynic effects [73]

When the study group was stratified by BMI, adults without obesity, those with class I obesity (BMI: 30-34.99 kg/m2) and those with class II obesity (BMI: 35-39.99 kg/m2) were less likely to have all-cause hospital admissions, ED visits, undergo an endoscopic procedure, receive a prescription for a prokinetic, and receive a laxative prescription if they used an incretin-based drug compared with nonusers

Then, the dose is increased every month or so, going up to 0.5 mg, then 1 mg, then 1.7 mg

For other biliary symptoms, temporarily holding the medication pending evaluation is appropriate