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The observed outcomes were particularly significant in fibroblast-like synoviocytes, key players in RA pathogenesis, with the following main effects: (1) inhibition of NF-B nuclear translocation, leading to a decrease in the production of pro-inflammatory cytokines, including TNF, IL-6, IL-8, IL-1, MCP-1, and High Mobility Group Box (HMGB)-1, all of which are involved in RA progression

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Mitochondrial ATP transporter Ant2 depletion impairs erythropoiesis and B lymphopoiesis

[DOI] [PubMed] [Google Scholar] 129.Coskun T., Urva S., Roell W.C., Qu H., Loghin C., Moyers J.S., OFarrell L.S., Briere D.A., Sloop K.W., Thomas M.K., et al
