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Available from: _Product_Information/human/002445/WC500140401.pdf

Ahrn B (2001) Inhibition of dipeptidyl peptidase-4 (DPP-4): a target to treat type 2 diabetes

As discussed above, lipopolysaccharides (LPS) could prompt inflammation and amyloidogenesis in the brain, by inducing the disorder of TNF-a, IL-1, and cyclooxygenase-2 (COX-2) (87)

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Mechanism of Action: GLP-1 receptor agonists work through multiple physiological pathways: Central appetite suppression : These medications cross the blood-brain barrier and activate GLP-1 receptors in the hypothalamus and brainstem, reducing hunger signals and increasing satiety Gastric emptying delay : By slowing stomach emptying, patients experience prolonged fullness after meals Insulin secretion enhancement : Glucose-dependent insulin release improves glycemic control Glucagon suppression : Reduced glucagon secretion decreases hepatic glucose production Tirzepatide offers dual agonism at both GLP-1 and glucose-dependent insulinotropic polypeptide (GIP) receptors, potentially providing enhanced weight loss efficacy compared to single-agonist medications