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To achieve a GLP-1RA that can act throughout the day, they made modifications and attached a fatty acid chain in the n-terminal that allows binding to albumin [19,20]
In experiments using animal models of nerve damage, the administration of GLP-1RA caused a significant increase in the rate of nerve regeneration
The authors hypothesized that the drug lowers migraine frequency by reducing intracranial pressure
By enhancing GLP-1 signaling, Teneligliptin helps reduce the expression of pro-inflammatory cytokines, thereby decreasing both peripheral and central nervous system inflammation [16]
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What if Im already taking GLP-1 medication
