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Anti-arteriosclerotic effects through GLP-1 receptor have been indicated so far only in basic experiments such as animal experimental model and/or cell culture system
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Accumulating evidence suggests that metabolic disturbances can promote the development of AF through structural and electrophysiological remodelling, but the underlying mechanisms that predispose an individual to AF are aetiology-dependent, thus emphasizing the need for tailored therapeutic strategies to treat AF that target an individuals metabolic profile
doi: 10.1210/jcem.87.3.8355 29 FabriciusTWVerhulstCEMKristensenPLTackCJMcCrimmonRJHellerSet al
PMCID: PMC12271809
Donega M, Fjordbakk CT, Kirk J, Sokal DM, Gupta I, Hunsberger GE, et al
