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Dendritic cell-based immunotherapy

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The mechanistic interpretation, debated for two decades, was that GIPR agonism did two things: it contributed to appetite suppression and energy expenditure in addition to GLP-1R activation, and - separately - it appeared to improve tolerability by reducing GLP-1-mediated nausea and GI adverse events

The medication works by stimulating appetite suppression in the brain and slowing stomach emptying, mechanisms completely separate from thyroid regulation